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Cai H, Harrison DG: Endothelial dysfunction in cardiovascular diseases: the role of oxidant stress

Somatic mutations in MED12, which are implicated in modulating oxidative stress in uterine fibroids, are believed to be reliable biomarkers for these tumors [6,7]

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Abstract Glucose-6-phosphate dehydrogenase (G6PD) deficiency, one of the most common human genetic enzymopathies, is caused by over 160 different point mutations and contributes to the severity of many acute and chronic diseases associated with oxidative stress, including hemolytic anemia and bilirubin-induced neurological damage particularly in newborns

N Engl J Med 2002